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Because of these and other kinds of evidence, the influence of environmental events on the frequency of stuttering is unquestionable. One theory assumes that children who stutter do not exhibit more dysfluencies than children who do not stutter. Behaviors that have been described as associated motor behaviors and avoidance responses are stuttering. Stuttering is all those actions a child takes not to stutter including the facial grimaces, arm swings, foot taps, tensed movements, and avoidance of words and speaking situations. What the child does to avoid normal non-fluencies is stuttering. Therefore, the child tries to avoid what the parents think is the problem which are the dysfluencies or normal non-fluencies. The problem of stuttering started after the mistaken diagnosis. One theory was that because of the misdiagnosis, the child believes that there is something wrong his or her speech. One theory was that because of the misdiagnosis, the child believes that believe that there is something wrong his or her speech. It was proposed that the origin of stuttering is its parental diagnosis and that stuttering is not in the mouth of the child but in the ear of the listener. A researcher had convinced that all speakers are dysfluent, and stuttering speakers are not necessarily more so. Parents who have very high standards of fluency, or those who do not understand that all children exhibit dysfluencies, diagnose stuttering in their normally fluent child who happens to repeat, interject, revise, and prolong segments of speech like most other children. A well-known theory of stuttering that explained it on the basis of environmental event is known as the diagnosogenic theory. It has been noted that stuttering varies depending on the listener. For example, in one study, a colored border placed on a printed page was associated with a high frequency of stuttering. Such movements are stutterings themselves. Another person, who was talking with relative fluency, walks into a fast-food place and cannot place an order. An individual who is talking fluently to a child turns to an adult and immediately stutters on the very first word. It already has been noted that stuttering varies depending on the listener. In essence, a colored border or a blotted out word may act as a new stimulus for new stuttering. In oral reading, when words that were stuttered on the first oral reading of a printed passage were all blotted out, the reader was likely to stutter on the words next to the blotted mark during the next reading. Later, the participants of the experiment again stuttered more in the presence of the colored border. A stimulus associated with increased or decreased stuttering tends to have the same effect on future occasions. For example, in one study, a colored border placed on a printed age was associated with a high frequency of stuttering. Many observations suggest that stuttering varies depending on environmental stimuli or events. That is why delayed feedback of speech induces more fluency because this delay now matches the built in delay. The auditory defect theory proposed that people who stutter have a built in delay in processing their own speech. These two findings have prompted speculation that the portion of the brain interprets one's own speech production and helps regulate it may be defective. These two findings have prompted speculation that the portion of the brain that interprets one's own speech production and thus helps regulate it may be defective. Much of the neurophysiological research describes the behavior of nerves and muscles of speech as they produce fluent and stuttered speech.
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1 Because of these and other kinds of evidence, the influence of environmental events on the frequency of stuttering is unquestionable. 2 One theory assumes that children who stutter do not exhibit more dysfluencies than children who do not stutter. 3 Behaviors that have been described as associated motor behaviors and avoidance responses are stuttering. 4 Stuttering is all those actions a child takes not to stutter including the facial grimaces, arm swings, foot taps, tensed movements, and avoidance of words and speaking situations. 5 What the child does to avoid normal non-fluencies is stuttering. 6 Therefore, the child tries to avoid what the parents think is the problem which are the dysfluencies or normal non-fluencies. 7 The problem of stuttering started after the mistaken diagnosis. 8 One theory was that because of the misdiagnosis, the child believes that there is something wrong his or her speech. 9 One theory was that because of the misdiagnosis, the child believes that believe that there is something wrong his or her speech. 10 It was proposed that the origin of stuttering is its parental diagnosis and that stuttering is not in the mouth of the child but in the ear of the listener. 11 A researcher had convinced that all speakers are dysfluent, and stuttering speakers are not necessarily more so. 12 Parents who have very high standards of fluency, or those who do not understand that all children exhibit dysfluencies, diagnose stuttering in their normally fluent child who happens to repeat, interject, revise, and prolong segments of speech like most other children. 13 A well-known theory of stuttering that explained it on the basis of environmental event is known as the diagnosogenic theory. 14 It has been noted that stuttering varies depending on the listener. 15 For example, in one study, a colored border placed on a printed page was associated with a high frequency of stuttering. 16 Such movements are stutterings themselves. 17 Another person, who was talking with relative fluency, walks into a fast-food place and cannot place an order. 18 An individual who is talking fluently to a child turns to an adult and immediately stutters on the very first word. 19 It already has been noted that stuttering varies depending on the listener. 20 In essence, a colored border or a blotted out word may act as a new stimulus for new stuttering. 21 In oral reading, when words that were stuttered on the first oral reading of a printed passage were all blotted out, the reader was likely to stutter on the words next to the blotted mark during the next reading. 22 Later, the participants of the experiment again stuttered more in the presence of the colored border. 23 A stimulus associated with increased or decreased stuttering tends to have the same effect on future occasions. 24 For example, in one study, a colored border placed on a printed age was associated with a high frequency of stuttering. 25 Many observations suggest that stuttering varies depending on environmental stimuli or events. 26 That is why delayed feedback of speech induces more fluency because this delay now matches the built in delay. 27 The auditory defect theory proposed that people who stutter have a built in delay in processing their own speech. 28 These two findings have prompted speculation that the portion of the brain interprets one's own speech production and helps regulate it may be defective. 29 These two findings have prompted speculation that the portion of the brain that interprets one's own speech production and thus helps regulate it may be defective. 30 Much of the neurophysiological research describes the behavior of nerves and muscles of speech as they produce fluent and stuttered speech.